Researchers have discovered how a cell surface protein called Aplp1 can play a role in spreading material responsible for Parkinson's disease from cell-to-cell in the brain.

During the pathogenesis of Parkinson’s disease (PD), α-synuclein pathology may originate in peripheral organs and spread to the CNS. Using human tissue samples and multiple approaches in mouse ...

In a recent study, talk therapy in Parkinson’s disease led to significant improvement in patients' well-being, ...

In the January 23 Nature Neuroscience, scientists led by Zhentao Zhang at Wuhan University, China, report that, in people with Parkinson’s and Lewy body diseases ... α-synuclein pathology can pass ...

Recent studies also showed that LBDs, particularly Parkinson's disease, often initiate in ... To identify the organs where α-synuclein pathology begins, we stained the peripheral organs and ...

New research uncovers how declining protein clearance mechanisms with age lead to Alzheimer’s, Parkinson’s, and other ...

Parkinson’s disease is a neurodegenerative disorder ... Joan Bennett, a professor of plant biology and pathology at Rutgers University since 2006, who is not involved in the lawsuit, spoke ...

Past studies on mice found Lag3 binds to alpha-synuclein proteins and spreads Parkinson's disease pathology in neurons. While deleting Lag3 significantly impedes this process, it does not ...